Abstract
Perinatal exposure to cadmium (Cd) or methylmercury (MeHg) results in impaired neurodevelopment. Thyroid hormone is essential for normal brain development. However, the issue whether Cd or MeHg, especially at low doses, interrupts thyroid hormone action remains to be investigated. In the present study, effects of perinatal exposure to low levels of Cd or MeHg on thyroid hormone metabolism were examined using metallothionein I and II (MT-I/II) null or wild-type neonatal mice.
OBJECTIVES: The impact of dental amalgam removal on the levels of anti- thyroid
peroxidase (anti-TPO) and anti-thyroglobulin (anti-Tg) antibodies was studied in
patients with autoimmune thyroiditis (AT) with and without mercury allergy.
METHODS: Thirty-nine patients with AT were tested by an optimized lymphocyte
proliferation test, MELISA® for allergy (hypersensitivity) to inorganic mercury.
Persons employed in high-risk lead-related occupations can transport lead dust home from a worksite through clothing, shoes, tools, or vehicles (1-4). During 2008, the Maine Childhood Lead Poisoning Prevention Program (MCLPPP) identified 55 new cases of elevated (≥15 µg/dL) venous blood lead levels (BLLs) among children aged <6 years through mandated routine screening (5,6). Although 90% of childhood lead poisoning cases in Maine during 2003–2007 had been linked to lead hazards in the child's home,
Objectives. We investigated whether foreign birthplace and residence were associated with an increased risk of childhood lead poisoning.
Methods. We conducted a matched case–control study among New York City children (mean age=3 years) tested for lead poisoning in 2002 (n=203 pairs). Children were matched on age, date of test,
