Inflammation plays a central role in the pathophysiology of atherosclerosis, starting from initiation, through progression, and ultimately the thrombotic complications of atherosclerosis. Diabetes mellitus is a major risk factor for atherosclerosis. Hyperglycemia-induced endothelial dysfunctions, along with hypercoagulable potential of diabetes mellitus, accelerate the process of atherothrombotic complications. Therefore, clinically feasible markers to monitor subtle systemic inflammatory burden and specific add-on for the same constitute need of the present day. The understanding of the concept of inflammation in diabetes-accelerated atherosclerosis can be used practically to predict future cardiovascular risk by evaluating inflammatory biomarkers and to design clinical trials making inflammation as a therapeutic target.
How Stem Cell Therapy Rejuvenates Hair Follicles and Promotes Growth
Hair thinning and hair loss often happen when follicles are no longer functioning at their best. Some follicles may slow
