Fat burning – Do I need a little help?
For centuries, people have been trying a wide variety of things to help to lose weight and decrease fat deposits on the body. Hundreds of ideas with diet and activities have been recommended and used, some fruitful yet others dangerous.
People typically follow eating patterns and diets based upon what is "handed" down from family and relatives, or from what friends or advertisers describe.
The "community" that should set standards, educate, and guide people on specifics in health, nutrition and diet has been the least helpful in this fight against fat. Of course this community refers to the medical establishment.
Until recent years, doctors never discussed nutrition with patients, nor did they describe medical research on how the body metabolizes fats, proteins and carbohydrates. In the majority of cases they still are going on archaic, useless information. The contrived idea of the food group patterns of eating has been wrong since its inception.
At any rate, a little specific knowledge, application and observation goes a very long way.
As I have seen it over the past 20 years, people do best with a lowered carbohydrate, moderate protein and low fat diet.
The parameters that control fat burning and weight loss are:
1. Carbohydrate intake
2. Exercise frequency and duration
3. Hormone levels
When carb intake is below 30 grams per day, most people start to burn cholesterol, triglycerides, excess blood sugar and as well lose weight. At levels between 30 to 50 grams per day people tend to stay at a steady state, and not gain or lose. Above and beyond 75 grams, to the hundreds, people gain.
Exercise should be done daily, and at the very least lasting 30 minutes. This could include walking, treadmill, bicycling etc.
Hormone levels may need to be done to assess:
– Growth Hormone
– Thyroid Hormone
– Adrenal Hormone
– Female Hormones
Correcting hormone deficiency can do a great deal to help burn fat.
As well, a not so new technique has emerged in the fight against fat – one that has been used internationally for over 50 years.
This is called Meso . In the 1950's Dr. Pistor in France created a technique to treat abnormalities of the skin.
Over many years and with varied solutions the has been used to treat many medical problems. The idea behind it is that solutions can be delivered into the skin with tiny injections and tiny needles to influence surrounding tissues. Such fluids can be more direct to the site and last longer than oral medications. These substances would not be "lost" to the entire body circulation and would be more effective.
They can be used for:
– Cellulite and fat burning
– Psoriasis and Dermatitis
– Muscloskeletal Disorders
– Pain Syndromes
– Shingles and Viral Skin Disorders
– Hair loss
and many others.
Relative to fat results, the solutions can be comprised of plant extracts, medications and homeopathics. They work in such a way to help stimulate Beta receptors on fat cells, inhibit Alpha receptors and create a breakdown and lysis of fat cells with a choline liquid. Millions of these injections have been done over the years with excellent results. Patients typically can lose an inch or so of fat in areas treated over the course of a week or two.
This technique is also important in that it can work on the areas that are the hardest areas to lose fat.
The best combination protocol involves the low carb diet, exercise, hormone correction and Meso .
See my website for low carb diet info, hormone evaluation and meso .
Dr. Chris Calapai
Histological changes associated with meso for fat dissolution.
Meso is a form of medical popular in Europe and South America. It is used for treating a variety of medical conditions, including the results of localized fat deposits and cellulite. Phosphatidylcholine/deoxycholate injections are a popular technique to treat localized fat accumulations and have recently become synonymous with meso , although their history and technique are distinct. To treat localized fat deposits, phosphatidylcholine (PC) and deoxycholate (DC) are utilized. To date, there have been no published histological studies that explain the mechanism of action of PC and DC.
Method. In this study the authors have obtained skin biopsies from a patient who had undergone meso with PC and DC. Punch biopsies were taken at one and two weeks after the procedure.
Results. Each of the biopsies taken at one and two weeks after results with PC and DC showed a normal epithelium and dermis, with a mixed septal and lobular panniculitis. The fat lobules were infiltrated by increased numbers of lymphocytes and, in particular, macrophages. The macrophages consisted of conventional forms, foam cells, and multinucleated fat-containing giant cells. The inflammation was associated with serous atrophy and microcyst formation.
Conclusion. This study demonstrates that meso with PC and DC affects the subcutaneous fat. We theorize that the reduction of subcutaneous fat likely follows inflammatory-mediated necrosis and resorption.
Phosphatidylcholine results to induce lipolysis.
The medicine Lipostabil N® has been in widespread use in Europe since 2002 by doctors working in the field of esthetics to achieve a reduction in the volume of smaller fat deposits by means of injections into the subcutaneous fatty tissue. The lipases released from the adipocytes by means of phosphatidylcholine produce a local breakdown of fat that is then discharged over the liver and metabolized via beta-oxidation. The medicine has been authorized for intravenous use in the prophylaxis and of fat embolisms and liver diseases.
The Effects of Low-Carbohydrate versus Conventional Weight Loss Diets in Severely Obese Adults: One-Year Follow-up of a Randomized Trial.
Background: A previous paper reported the 6-month comparison of weight loss and metabolic changes in obese adults randomly assigned to either a low-carbohydrate diet or a conventional weight loss diet.
Objective: To review the 1-year outcomes between these diets.
Design: Randomized trial.
Setting: Philadelphia Veterans Affairs Medical Center.
Participants: 132 obese adults with a body mass index of 35 kg/m2 or greater; 83% had diabetes or the metabolic syndrome.
Intervention: Participants received counseling to either restrict carbohydrate intake to <30 g per day (low-carbohydrate diet) or to restrict caloric intake by 500 calories per day with <30% of calories from fat (conventional diet).
Measurements: Changes in weight, lipid levels, glycemic control, and insulin sensitivity.
Results: By 1 year, mean (±SD) weight change for persons on the low-carbohydrate diet was –5.1 ± 8.7 kg compared with –3.1 ± 8.4 kg for persons on the conventional diet. Differences between groups were not significant (–1.9 kg [95% CI, –4.9 to 1.0 kg]; P = 0.20). For persons on the low-carbohydrate diet, triglyceride levels decreased more (P = 0.044) and high-density lipoprotein cholesterol levels decreased less (P = 0.025). As seen in the small group of persons with diabetes (n = 54) and after adjustment for covariates, hemoglobin A1c levels improved more for persons on the low-carbohydrate diet. These more favorable metabolic responses to a low-carbohydrate diet remained significant after adjustment for weight loss differences. Changes in other lipids or insulin sensitivity did not differ between groups.
Limitations: These findings are limited by a high dropout rate (34%) and by suboptimal dietary adherence of the enrolled persons.
Conclusion: Participants on a low-carbohydrate diet had more favorable overall outcomes at 1 year than did those on a conventional diet. Weight loss was similar between groups, but effects on atherogenic dyslipidemia and glycemic control were still more favorable with a low-carbohydrate diet after adjustment for differences in weight loss.